Good outcome of surgical splenectomy in the presence of pancytopenia in an adult sickle cell anaemia patient with splenic sequestration and massive splenomegaly
PDF

Keywords

Ppancytopenia
sickle cell anaemia
splenic sequestration
surgical splenectomy

Abstract

Background: Sickle cell disease (SCD) are genetic

diseases of the red blood cell resulting from the

presence of a mutated form of haemoglobin,

haemoglobin S (HbS). Splenic sequestration is a life

threatening complication seen commonly in children

with SCA.

Method: We present a female adult patient with SCA

who had massive splenomegaly and pancytopenia due

to splenic sequestration and hypersplenism to whom

successful surgical splenectomy was done in the

presence of pancytopenia. The aim of this report was

to make known to health workers that splenectomy

can be done successfully in life-threatening splenic

sequestration in the presence of pancytopenia with

multidisciplinary collaboration. Miss B is a 27yr old

SCA patient who was referred to our center from a

peripheral hospital with complaint of abdominal

distension for about two years, abdominal pain for

about one month and rib pain for three days prior to

presentation. Abdominal pain was dull in character

and associated with abdominal swelling. There was

associated easy satiety, weight loss, yellowness of

the eyes, passage of coke-coloured urine, dizziness,

weakness and breathlessness on mild exertion.

Result: Examination showed a young woman in no

obvious distress, afebrile (temperature 36.70C), mildly

icteric, pale and dehydrated. Vital signs were normal.

Examination of the chest showed no abnormality.

Abdominal examination showed hepatomegaly of

10cm below the right costal margin and splenomegaly

of 26cm below the left costal margin. Laboratory

investigation showed anaemia with haemoglobin level

of 4.1g/dl, white blood cell and platelet count wer

within normal range. Repeated blood transfusion was

given and the spleen size progressively increased in

size with resultant pancytopenia (haemoglobin level

of 2.9g/dl, white blood cell count of 2.2 X 109 /l and

platelet count of 41 X 109 /l).

Conclusion: Surgical splenectomy was subsequently

done successfully in the presence of pancytopenia

by the collaboration of a team of Haematologist,

Surgeon and Anaesthetist. Post–operatively,

haematological parameters improved and patient was

discharged. Successful surgical splenectomy can be

done in the presence of pancytopenia.

Interdisciplinary collaboration is key to successful

outcome in the management of such complication.

PDF

References

Hsieh MM, Tisdale JF and Rodgers GP. Hemolytic

Anemia: Thalassemias and Sickle Cell Disorders.

In: Rodgers GP, Young NS (editors). Bethesda

Handbook of Clinical Hematology. 2nd ed.

Philadelphia; Lippincott Williams & Wilkins: 2010

p35 – 55.

Serjeant GR. Sickle-cell disease The Lancet 1997;

(9079): 725–730.

Modell B and Darlison M. Global epidemiology

of haemoglobin disorders and derived service

indicators. Bulletin of the World Health

Organization 2008; 86 (6):480–487

Isa HA, Adegoke SA, Madu AJ, et al. Sickle

Cell Disease Clinical Phenotypes in Nigeria: A

preliminary analysis of the Sickle pan Africa

Research consortium (SPARCO) Nigeria

Database. Blood cells, Molecules and Diseases

; 84: 102438.

Ugwu NI, Nna EO, Ugwu CN, et al. Evaluation

of Fetal Haemoglobin status among Nigerian

patients with sickle cell anaemia using High

Performance Liquid Chromatography. West

African Journal of Medicine 2021; 38(3): 222 –

Ali Al-Barazanchi ZA, Abdulateef SS and Hassan

MK. Co-inheritance of Ü-thalassaemia gene

mutation in patients with sickle cell Disease:

impact on clinical and haematological variables.

Niger J Clin Pract 2021; 24: 874 – 882.

Tewari S, Brousse V, Piel FB, Menzel S and

Rees DC. Environmental determinants of

severity in sickle cell disease. Haematologica.

;100(9):1108-1116.

Olabode JO and Shokunbi WA. Types of crises

in sickle cell disease patients presenting at the

haematology day care unit (HDCU), University

College Hospital (UCH), Ibadan. West Afr J

Med. 2006 Oct-Dec; 25(4):284-288.

Brousse V, Buffet P and Rees D. The spleen and

sickle cell Disease: the sick(led) spleen. British

Journal of Haematology 2014; 166: 165 – 176..

Ladu AI, Aiyenigba AO, Adekile A and Bates I.

The spectrum of splenic complications in patients

with sickle cell disease in Africa: a systematic

review. British Journal of Haematology 2021;

(1): 26 – 42.

Durosinmi MA, Salawu L, Ova YA, Lawal OO

and Fadiran OA. Haematological parameters in

sickle cell anaemia patients with and without

splenomegaly. Niger Postgrad Med J 2005: 12(4):

– 274.

Crosby LE, Quinn CT and Kalinyak KA. A

biophstchosocial model for the management of

patients with sickle-cell disease transitioning to

adult medical care. Advances in Therapy 2015;

: 293 – 305.

Bisharat N, Omari H, Lavi I and Raz R. Risk of

infection and death among post-splenectomy

patients. J Infect. 2001 Oct;43(3):182-186

Luu S, Spelman D and Woolley D. Postsplenectomy sepsis: preventive strategies,

challenges and solutions. Infection and Drug

Resistance 2019; 12: 2839 – 2851.

Lee GM. Preventing infections in children and adults

with asplenia. Hematology 2020; 4: 328 – 335.